Short-term venous stasis induces fibrinolytic activation but not thrombin formation

Rühl H, Müller J, Wäschenbach J, Oldenburg J, Pötzsch B, Dewald O (2014)

Publication Type: Journal article

Publication year: 2014

Journal

Journal of Atherosclerosis and Thrombosis Japan Atherosclerosis Society

Book Volume: 21

Pages Range: 1260-1270

Journal Issue: 12

DOI: 10.5551/jat.24216

Abstract

Aim: Venous stasis is a well-known risk factor for the development of venous thromboembolism. It is likely that stasis increases the risk of thrombosis by inducing hypercoagulability via the hypoxic procoagulant activation of endothelial and mononuclear cells and the accumulation of activated clotting factors. However, increased rates of thrombin formation have not been demonstrated in response to venous stasis in vivo.

Authors with CRIS profile

Oliver Dewald

Involved external institutions

Universitätsklinikum Bonn DE Germany (DE)

How to cite

APA:

Rühl, H., Müller, J., Wäschenbach, J., Oldenburg, J., Pötzsch, B., & Dewald, O. (2014). Short-term venous stasis induces fibrinolytic activation but not thrombin formation. Journal of Atherosclerosis and Thrombosis, 21(12), 1260-1270. https://doi.org/10.5551/jat.24216

MLA:

Rühl, Heiko, et al. "Short-term venous stasis induces fibrinolytic activation but not thrombin formation." Journal of Atherosclerosis and Thrombosis 21.12 (2014): 1260-1270.

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