Moese S, Selbach M, Meyer TF, Backert S (2002)
Publication Status: Published
Publication Type: Journal article
Publication year: 2002
Book Volume: 70
Pages Range: 4687-91
Journal Issue: 8
DOI: 10.1128/iai.70.8.4687-4691.2002
Infection with cag+ but not cag-negative Helicobacter pylori leads to the formation of large homotypic aggregates of macrophage-like cells. Intracellular adhesion molecule 1 is up-regulated and recruited to the cell surface of infected cells and mediates the aggregation via lymphocyte function-associated molecule 1. This signaling may regulate cell-cell interactions and inflammatory responses.
APA:
Moese, S., Selbach, M., Meyer, T.F., & Backert, S. (2002). cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface. Infection and Immunity, 70(8), 4687-91. https://doi.org/10.1128/iai.70.8.4687-4691.2002
MLA:
Moese, S., et al. "cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface." Infection and Immunity 70.8 (2002): 4687-91.
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