Home Publications Research Grants Inventions & Patents Awards Additional Research Activities Faculties & Institutions Research Areas

cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface.

Moese S, Selbach M, Meyer TF, Backert S (2002)

---

Publication Status: Published

Publication Type: Journal article

Publication year: 2002

Journal

Book Volume: 70

Pages Range: 4687-91

Journal Issue: 8

DOI: 10.1128/iai.70.8.4687-4691.2002

Abstract

Infection with cag+ but not cag-negative Helicobacter pylori leads to the formation of large homotypic aggregates of macrophage-like cells. Intracellular adhesion molecule 1 is up-regulated and recruited to the cell surface of infected cells and mediates the aggregation via lymphocyte function-associated molecule 1. This signaling may regulate cell-cell interactions and inflammatory responses.

Authors with CRIS profile

How to cite

APA:

Moese, S., Selbach, M., Meyer, T.F., & Backert, S. (2002). cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface. Infection and Immunity, 70(8), 4687-91. https://doi.org/10.1128/iai.70.8.4687-4691.2002

MLA:

Moese, S., et al. "cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface." Infection and Immunity 70.8 (2002): 4687-91.

BibTeX: Download