Willam C (2014)
Publication Type: Journal article, Note
Publication year: 2014
Book Volume: 85
Pages Range: 232-4
Journal Issue: 2
DOI: 10.1038/ki.2013.362
An inflammatory stimulus prior to an ischemic insult can be protective in acute kidney injury (AKI) as well as other acute organ injury models, an effect called cross-tolerance. He et al. investigated mechanisms of cross-tolerance whereby pretreatment with lipopolysaccharide (LPS) protects from a subsequent ischemic insult of the kidney. The protection was mediated by LPS-induced nuclear factor-?B and hypoxia-inducible factor-2? (HIF-2?) signaling. These results link two central cellular pathways and give new insight into HIF-mediated renoprotection in AKI models.
APA:
Willam, C. (2014). HIF meets NF-?B signaling. Kidney International, 85(2), 232-4. https://doi.org/10.1038/ki.2013.362
MLA:
Willam, Carsten. "HIF meets NF-?B signaling." Kidney International 85.2 (2014): 232-4.
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