apl. Prof. Dr. Elisabeth Sock

Medizinische Fakultät Lehrstuhl für Biochemie und Pathobiochemie

Replacement of related POU transcription factors leads to severe defects in mouse forebrain development. (2009) Sock E, Reiprich S, Müller J, Stolt C, Wegner M Journal article, Original article Sox12 deletion in the mouse reveals non-reciprocal redundancy with the related Sox4 and Sox11 transcription factors. (2008) Wegner M, Sock E Journal article, Original article Anterior segment dysgenesis in the eyes of mice deficient for the high-mobility-group transcription factor Sox11. (2008) Sock E, Wegner M Journal article, Original article Prolonged glial expression of Sox4 in the central nervous system leads to architectural cerebellar defects and ataxia. (2007) Sock E, Wegner M Journal article, Original article SoxD proteins influence multiple stages of oligodendrocyte development and modulate SoxE protein function. (2006) Sock E, Wegner M, Stolt C Journal article, Original article Impact of transcription factor Sox8 on oligodendrocyte specification in the mouse embryonic spinal cord. (2005) Sock E, Wegner M, Stolt C Journal article, Original article The high-mobility-group transcription factor Sox8 is a negative regulator of osteoblast differentiation. (2005) Sock E, Wegner M Journal article, Original article The POU protein Oct-6 is a nucleocytoplasmic shuttling protein. (2005) Sock E, Wegner M Journal article, Original article Gene targeting reveals a widespread role for the high-mobility-group transcription factor Sox11 in tissue remodeling. (2004) Sock E, Wegner M Journal article, Original article The Sox9 transcription factor determines glial fate choice in the developing spinal cord. (2003) Sock E, Wegner M, Stolt C Journal article, Original article