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Prof. Dr. Christoph Korbmacher
List of publications:
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Lehrstuhl für Physiologie (Vegetative Physiologie)
Project Leads
(9)
Publications
(74)
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Journal article
Journal article
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Book chapter / Article in edited volumes
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Authored book
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Translation
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Thesis
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Conference contribution
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Erratum to: Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase. (2017)
Sostegni S, Diakov A, Mcintyre P, Bunnett N, Korbmacher C, Härteis S
Journal article, Erratum
Bile acids potentiate proton-activated currents in Xenopus laevis oocytes expressing human acid-sensing ion channel (ASIC1a). (2017)
Ilyaskin A, Diakov A, Korbmacher C, Härteis S
Journal article
Lithocholic acid has a sustained stimulatory effect on the epithelial sodium channel (ENaC) in mouse late distal colon (2017)
Bertog M, Reeh L, Bunnett NW, Korbmacher C
Conference contribution
Activation of the Epithelial Sodium Channel (ENaC) by Aldosterone in mCCD(cl1) Mouse Renal Cortical Collecting Duct Cells is Dependent on SGK1 and Can be Prevented by Inhibiting Endogenous Proteases (2017)
Korbmacher C, Mansley M, Bertog M
Conference contribution
δβγ-ENaC is inhibited by CFTR but stimulated by cAMP in Xenopus laevis oocytes (2017)
Rauh R, Hörner C, Korbmacher C
Journal article, Original article
In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron (2016)
Nesterov V, Krüger B, Bertog M, Dahlmann A, Palmisano R, Korbmacher C
Journal article
Activation of the Human Epithelial Sodium Channel (ENaC) by Bile Acids Involves the Degenerin Site (2016)
Ilyaskin A, Diakov A, Korbmacher C, Härteis S
Journal article
Association of Plasminuria with Overhydration in Patients with CKD (2016)
Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Härteis S, et al.
Journal article
mTORC2 critically regulates renal potassium handling (2016)
Grahammer F, Nesterov V, Ahmed A, Steinhardt F, Sandner L, Arnold F, Cordts T, et al.
Journal article
Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain (2015)
Zhao P, Lieu T, Barlow N, Sostegni S, Härteis S, Korbmacher C, Liedtke W, et al.
Journal article
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