Prof. Dr. Christoph Korbmacher

Lehrstuhl für Physiologie (Vegetative Physiologie)

Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome (2019) Bohnert BN, Daiminger S, Woern M, Sure F, Staudner T, Ilyaskin A, Batbouta F, et al. Journal article Rebuttal to editorial: Sodium retention by uPA in nephrotic syndrome? (2019) Bohnert BN, Kanse S, Härteis S, Korbmacher C, Artunc F Journal article The phosphorylation site T613 in the β-subunit of rat epithelial Na+channel (ENaC) modulates channel inhibition by Nedd4-2 (2018) Krüger B, Yang L, Korbmacher C, Rauh R Journal article The degenerin region of the human bile acid-sensitive ion channel (BASIC) is involved in channel inhibition by calcium and activation by bile acids. (2018) Ilyaskin A, Kirsch S, Böckmann R, Sticht H, Korbmacher C, Härteis S, Diakov A Journal article, Original article Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice. (2018) Härteis S, Schork A, Doerffel T, Bohnert BN, Nacken R, Woern M, Xiao M, et al. Journal article Inhibitors of the proteasome stimulate the epithelial sodium channel (ENaC) through SGK1 and mimic the effect of aldosterone (2018) Mansley M, Korbmacher C, Bertog M Journal article Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome (2018) Bohnert BN, Menacher M, Janessa A, Woern M, Schork A, Daiminger S, Kalbacher H, et al. Journal article Erratum to: Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase. (2017) Sostegni S, Diakov A, Mcintyre P, Bunnett N, Korbmacher C, Härteis S Journal article, Erratum Bile acids potentiate proton-activated currents in Xenopus laevis oocytes expressing human acid-sensing ion channel (ASIC1a). (2017) Ilyaskin A, Diakov A, Korbmacher C, Härteis S Journal article Lithocholic acid has a sustained stimulatory effect on the epithelial sodium channel (ENaC) in mouse late distal colon (2017) Bertog M, Reeh L, Bunnett NW, Korbmacher C Conference contribution