Silke Härteis



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Book chapter / Article in edited volumes
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Unpublished / Preprint

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Journal

Rebuttal to editorial: Sodium retention by uPA in nephrotic syndrome? (2019) Bohnert BN, Kanse S, Härteis S, Korbmacher C, Artunc F Journal article The degenerin region of the human bile acid-sensitive ion channel (BASIC) is involved in channel inhibition by calcium and activation by bile acids. (2018) Ilyaskin A, Kirsch S, Böckmann R, Sticht H, Korbmacher C, Härteis S, Diakov A Journal article, Original article Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice. (2018) Härteis S, Schork A, Doerffel T, Bohnert BN, Nacken R, Woern M, Xiao M, et al. Journal article Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome (2018) Bohnert BN, Menacher M, Janessa A, Woern M, Schork A, Daiminger S, Kalbacher H, et al. Journal article Erratum to: Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase. (2017) Sostegni S, Diakov A, Mcintyre P, Bunnett N, Korbmacher C, Härteis S Journal article, Erratum Bile acids potentiate proton-activated currents in Xenopus laevis oocytes expressing human acid-sensing ion channel (ASIC1a). (2017) Ilyaskin A, Diakov A, Korbmacher C, Härteis S Journal article Activation of the Human Epithelial Sodium Channel (ENaC) by Bile Acids Involves the Degenerin Site (2016) Ilyaskin A, Diakov A, Korbmacher C, Härteis S Journal article Association of Plasminuria with Overhydration in Patients with CKD (2016) Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Härteis S, et al. Journal article Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain (2015) Zhao P, Lieu T, Barlow N, Sostegni S, Härteis S, Korbmacher C, Liedtke W, et al. Journal article Atomic force microscopy imaging reveals the formation of ASIC/ENaC cross-clade ion channels (2015) Jeggle P, Smith ESJ, Stewart AP, Härteis S, Korbmacher C, Edwardson JM Journal article
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