Department of Medicine 1 – Gastroenterology, Pneumology and Endocrinology


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Types of publications

Journal article
Book chapter / Article in edited volumes
Authored book
Translation
Thesis
Edited Volume
Conference contribution
Other publication type
Unpublished / Preprint

Publication year

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To

Abstract

Journal

Non-professional phagocytosis: a general feature of normal tissue cells (2019) Seeberg J, Loibl M, Moser F, Schwegler M, Büttner-Herold M, Daniel C, Engel F, et al. Journal article, Original article What are new treatment concepts in systemic itch? (2019) Kremer A Journal article, Review article Ethanol consumption decreases autoantibody titers by blocking IL-21 secretion of Tfh cells during the development of arthritis (2019) Azizov V, Dietel K, Kolenbrander A, Seubert S, Steffen F, Meidenbauer J, Steffen U, et al. Conference contribution The immunological role of adipose tissue in inflammatory bowel disease (2019) Letizia M, Boettcher C, Sillke YR, Schmidt F, Günther C, Kunkel D, Glauben R, et al. Conference contribution Tumor immunoevasion by high glycolytic tumors through an acidosis-dependent induction of noninflammatory tumor-associated macrophages (2019) Bohn T, Rapp S, Pektor S, Gerlach K, Weigmann B, Lueckel C, Truong N, et al. Conference contribution Diminished allergen-induced intestinal and airway inflammation in humanized mice by depletion of natural killer cells prior to PBMC engraftment (2019) Ose R, Weigmann B, Maxeiner J, Schuster P, Saloga J, Bellinghausen I Conference contribution The role of the chemokine receptor CCR8 in migration and effector function of innate lymphoid cells type 2 (2019) Knipfer L, Schulz-Kuhnt A, Symowski C, Vöhringer D, Neurath M, Atreya I, Wirtz S Conference contribution Functional role of the Tec kinase ITK in the pathogenesis of inflammatory bowel disease (2019) Lechner K, Neurath M, Weigmann B Conference contribution The role of IRF-1 during pathogen-induced intestinal Inflammation (2019) Schmalzl A, Neurath M, Wirtz S Conference contribution Acetaminophen-induced liver injury is mediated by the ion channel TRPV4 (2019) Echtermeyer F, Eberhardt M, Risser L, Herzog C, Gueler F, Khalil M, Engel M, et al. Journal article